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Excessive microglial activation aggravates olfactory dysfunction by impeding the survival of newborn neurons in the olfactory bulb of Niemann-Pick disease type C1 mice.

Biochimica et biophysica acta

Seo Y, Kim HS, Shin Y, Kang I, Choi SW, Yu KR, Seo KW, Kang KS.
PMID: 25132229
Biochim Biophys Acta. 2014 Nov;1842(11):2193-203. doi: 10.1016/j.bbadis.2014.08.005. Epub 2014 Aug 15.

Progressive olfactory impairment is one of the earliest markers of neurodegeneration. However, the underlying mechanism for this dysfunction remains unclear. The present study investigated the possible role of microgliosis in olfactory deficits using a mouse model of Niemann-Pick disease...

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Seo Y, Kim HS, Shin Y, et al. Excessive microglial activation aggravates olfactory dysfunction by impeding the survival of newborn neurons in the olfactory bulb of Niemann-Pick disease type C1 mice. Biochim Biophys Acta. 2014;1842(11):2193-203doi: 10.1016/j.bbadis.2014.08.005.
Seo, Y., Kim, H. S., Shin, Y., Kang, I., Choi, S. W., Yu, K. R., Seo, K. W., & Kang, K. S. (2014). Excessive microglial activation aggravates olfactory dysfunction by impeding the survival of newborn neurons in the olfactory bulb of Niemann-Pick disease type C1 mice. Biochimica et biophysica acta, 1842(11), 2193-203. https://doi.org/10.1016/j.bbadis.2014.08.005
Seo, Yoojin, et al. "Excessive microglial activation aggravates olfactory dysfunction by impeding the survival of newborn neurons in the olfactory bulb of Niemann-Pick disease type C1 mice." Biochimica et biophysica acta vol. 1842,11 (2014): 2193-203. doi: https://doi.org/10.1016/j.bbadis.2014.08.005
Seo Y, Kim HS, Shin Y, Kang I, Choi SW, Yu KR, Seo KW, Kang KS. Excessive microglial activation aggravates olfactory dysfunction by impeding the survival of newborn neurons in the olfactory bulb of Niemann-Pick disease type C1 mice. Biochim Biophys Acta. 2014 Nov;1842(11):2193-203. doi: 10.1016/j.bbadis.2014.08.005. Epub 2014 Aug 15. PMID: 25132229.
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Blockade of exosome generation with GW4869 dampens the sepsis-induced inflammation and cardiac dysfunction.

Biochimica et biophysica acta

Essandoh K, Yang L, Wang X, Huang W, Qin D, Hao J, Wang Y, Zingarelli B, Peng T, Fan GC.
PMID: 26300484
Biochim Biophys Acta. 2015 Nov;1852(11):2362-71. doi: 10.1016/j.bbadis.2015.08.010. Epub 2015 Aug 20.

Sepsis is an infection-induced severe inflammatory disorder that leads to multiple organ failure. Amongst organs affected, myocardial depression is believed to be a major contributor to septic death. While it has been identified that large amounts of circulating pro-inflammatory...

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Essandoh K, Yang L, Wang X, et al. Blockade of exosome generation with GW4869 dampens the sepsis-induced inflammation and cardiac dysfunction. Biochim Biophys Acta. 2015;1852(11):2362-71doi: 10.1016/j.bbadis.2015.08.010.
Essandoh, K., Yang, L., Wang, X., Huang, W., Qin, D., Hao, J., Wang, Y., Zingarelli, B., Peng, T., & Fan, G. C. (2015). Blockade of exosome generation with GW4869 dampens the sepsis-induced inflammation and cardiac dysfunction. Biochimica et biophysica acta, 1852(11), 2362-71. https://doi.org/10.1016/j.bbadis.2015.08.010
Essandoh, Kobina, et al. "Blockade of exosome generation with GW4869 dampens the sepsis-induced inflammation and cardiac dysfunction." Biochimica et biophysica acta vol. 1852,11 (2015): 2362-71. doi: https://doi.org/10.1016/j.bbadis.2015.08.010
Essandoh K, Yang L, Wang X, Huang W, Qin D, Hao J, Wang Y, Zingarelli B, Peng T, Fan GC. Blockade of exosome generation with GW4869 dampens the sepsis-induced inflammation and cardiac dysfunction. Biochim Biophys Acta. 2015 Nov;1852(11):2362-71. doi: 10.1016/j.bbadis.2015.08.010. Epub 2015 Aug 20. PMID: 26300484; PMCID: PMC4581992.
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Expansion size and presence of CCG/CTC/CGG sequence interruptions in the expanded CTG array are independently associated to hypermethylation at the DMPK locus in myotonic dystrophy type 1 (DM1).

Biochimica et biophysica acta

Santoro M, Fontana L, Masciullo M, Bianchi ML, Rossi S, Leoncini E, Novelli G, Botta A, Silvestri G.
PMID: 26391753
Biochim Biophys Acta. 2015 Dec;1852(12):2645-52. doi: 10.1016/j.bbadis.2015.09.007. Epub 2015 Sep 21.

No abstract available.

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Santoro M, Fontana L, Masciullo M, et al. Expansion size and presence of CCG/CTC/CGG sequence interruptions in the expanded CTG array are independently associated to hypermethylation at the DMPK locus in myotonic dystrophy type 1 (DM1). Biochim Biophys Acta. 2015;1852(12):2645-52doi: 10.1016/j.bbadis.2015.09.007.
Santoro, M., Fontana, L., Masciullo, M., Bianchi, M. L., Rossi, S., Leoncini, E., Novelli, G., Botta, A., & Silvestri, G. (2015). Expansion size and presence of CCG/CTC/CGG sequence interruptions in the expanded CTG array are independently associated to hypermethylation at the DMPK locus in myotonic dystrophy type 1 (DM1). Biochimica et biophysica acta, 1852(12), 2645-52. https://doi.org/10.1016/j.bbadis.2015.09.007
Santoro, Massimo, et al. "Expansion size and presence of CCG/CTC/CGG sequence interruptions in the expanded CTG array are independently associated to hypermethylation at the DMPK locus in myotonic dystrophy type 1 (DM1)." Biochimica et biophysica acta vol. 1852,12 (2015): 2645-52. doi: https://doi.org/10.1016/j.bbadis.2015.09.007
Santoro M, Fontana L, Masciullo M, Bianchi ML, Rossi S, Leoncini E, Novelli G, Botta A, Silvestri G. Expansion size and presence of CCG/CTC/CGG sequence interruptions in the expanded CTG array are independently associated to hypermethylation at the DMPK locus in myotonic dystrophy type 1 (DM1). Biochim Biophys Acta. 2015 Dec;1852(12):2645-52. doi: 10.1016/j.bbadis.2015.09.007. Epub 2015 Sep 21. PMID: 26391753.
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The role of sphingosine-1-phosphate in endothelial barrier function.

Biochimica et biophysica acta

Wilkerson BA, Argraves KM.
PMID: 25009123
Biochim Biophys Acta. 2014 Oct;1841(10):1403-1412. doi: 10.1016/j.bbalip.2014.06.012. Epub 2014 Jul 05.

Loss of endothelial barrier function is implicated in the etiology of metastasis, atherosclerosis, sepsis and many other diseases. Studies suggest that sphingosine-1-phosphate (S1P), particularly HDL-bound S1P (HDL-S1P) is essential for endothelial barrier homeostasis and that HDL-S1P may be protective...

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Wilkerson BA, Argraves KM. The role of sphingosine-1-phosphate in endothelial barrier function. Biochim Biophys Acta. 2014;1841(10):1403-1412doi: 10.1016/j.bbalip.2014.06.012.
Wilkerson, B. A., & Argraves, K. M. (2014). The role of sphingosine-1-phosphate in endothelial barrier function. Biochimica et biophysica acta, 1841(10), 1403-1412. https://doi.org/10.1016/j.bbalip.2014.06.012
Wilkerson, Brent A, and Argraves, Kelley M. "The role of sphingosine-1-phosphate in endothelial barrier function." Biochimica et biophysica acta vol. 1841,10 (2014): 1403-1412. doi: https://doi.org/10.1016/j.bbalip.2014.06.012
Wilkerson BA, Argraves KM. The role of sphingosine-1-phosphate in endothelial barrier function. Biochim Biophys Acta. 2014 Oct;1841(10):1403-1412. doi: 10.1016/j.bbalip.2014.06.012. Epub 2014 Jul 05. PMID: 25009123; PMCID: PMC4169319.
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Bcl3 regulates pro-survival and pro-inflammatory gene expression in cutaneous T-cell lymphoma.

Biochimica et biophysica acta

Chang TP, Vancurova I.
PMID: 25089799
Biochim Biophys Acta. 2014 Nov;1843(11):2620-30. doi: 10.1016/j.bbamcr.2014.07.012. Epub 2014 Jul 30.

The advanced stages of cutaneous T cell lymphoma (CTCL) are characterized not only by decreased levels of pro-inflammatory cytokines, resulting in high susceptibility to infections, but also by high constitutive activity of NFκB, which promotes cell survival and resistance...

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Chang TP, Vancurova I. Bcl3 regulates pro-survival and pro-inflammatory gene expression in cutaneous T-cell lymphoma. Biochim Biophys Acta. 2014;1843(11):2620-30doi: 10.1016/j.bbamcr.2014.07.012.
Chang, T. P., & Vancurova, I. (2014). Bcl3 regulates pro-survival and pro-inflammatory gene expression in cutaneous T-cell lymphoma. Biochimica et biophysica acta, 1843(11), 2620-30. https://doi.org/10.1016/j.bbamcr.2014.07.012
Chang, Tzu-Pei, and Vancurova, Ivana. "Bcl3 regulates pro-survival and pro-inflammatory gene expression in cutaneous T-cell lymphoma." Biochimica et biophysica acta vol. 1843,11 (2014): 2620-30. doi: https://doi.org/10.1016/j.bbamcr.2014.07.012
Chang TP, Vancurova I. Bcl3 regulates pro-survival and pro-inflammatory gene expression in cutaneous T-cell lymphoma. Biochim Biophys Acta. 2014 Nov;1843(11):2620-30. doi: 10.1016/j.bbamcr.2014.07.012. Epub 2014 Jul 30. PMID: 25089799; PMCID: PMC4158825.
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Helicase associated 2 domain is essential for helicase activity of RNA helicase A.

Biochimica et biophysica acta

Xing L, Zhao X, Niu M, Kleiman L.
PMID: 25062910
Biochim Biophys Acta. 2014 Oct;1844(10):1757-64. doi: 10.1016/j.bbapap.2014.07.001. Epub 2014 Jul 23.

RNA helicase A (RHA), a DExD/H box protein, plays critical roles in a wide variety of cellular or viral functions. RHA contains a conserved core helicase domain that is flanked by five other domains. Two double-stranded RNA binding domains...

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Xing L, Zhao X, Niu M, et al. Helicase associated 2 domain is essential for helicase activity of RNA helicase A. Biochim Biophys Acta. 2014;1844(10):1757-64doi: 10.1016/j.bbapap.2014.07.001.
Xing, L., Zhao, X., Niu, M., & Kleiman, L. (2014). Helicase associated 2 domain is essential for helicase activity of RNA helicase A. Biochimica et biophysica acta, 1844(10), 1757-64. https://doi.org/10.1016/j.bbapap.2014.07.001
Xing, Li, et al. "Helicase associated 2 domain is essential for helicase activity of RNA helicase A." Biochimica et biophysica acta vol. 1844,10 (2014): 1757-64. doi: https://doi.org/10.1016/j.bbapap.2014.07.001
Xing L, Zhao X, Niu M, Kleiman L. Helicase associated 2 domain is essential for helicase activity of RNA helicase A. Biochim Biophys Acta. 2014 Oct;1844(10):1757-64. doi: 10.1016/j.bbapap.2014.07.001. Epub 2014 Jul 23. PMID: 25062910.
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Structure and properties of the catalytic site of nitric oxide reductase at ambient temperature.

Biochimica et biophysica acta

Daskalakis V, Ohta T, Kitagawa T, Varotsis C.
PMID: 26140941
Biochim Biophys Acta. 2015 Oct;1847(10):1240-4. doi: 10.1016/j.bbabio.2015.06.014. Epub 2015 Jul 02.

Nitric oxide reductase (Nor) is the third of the four enzymes of bacterial denitrification responsible for the catalytic formation of laughing gas (N2O). Here we report the detection of the hyponitrite (HO-N=N-O(-)) species (νN-N=1332cm(-1)) in the heme b3 Fe-FeB...

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Daskalakis V, Ohta T, Kitagawa T, et al. Structure and properties of the catalytic site of nitric oxide reductase at ambient temperature. Biochim Biophys Acta. 2015;1847(10):1240-4doi: 10.1016/j.bbabio.2015.06.014.
Daskalakis, V., Ohta, T., Kitagawa, T., & Varotsis, C. (2015). Structure and properties of the catalytic site of nitric oxide reductase at ambient temperature. Biochimica et biophysica acta, 1847(10), 1240-4. https://doi.org/10.1016/j.bbabio.2015.06.014
Daskalakis, Vangelis, et al. "Structure and properties of the catalytic site of nitric oxide reductase at ambient temperature." Biochimica et biophysica acta vol. 1847,10 (2015): 1240-4. doi: https://doi.org/10.1016/j.bbabio.2015.06.014
Daskalakis V, Ohta T, Kitagawa T, Varotsis C. Structure and properties of the catalytic site of nitric oxide reductase at ambient temperature. Biochim Biophys Acta. 2015 Oct;1847(10):1240-4. doi: 10.1016/j.bbabio.2015.06.014. Epub 2015 Jul 02. PMID: 26140941.
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Induced pluripotency enables differentiation of human nullipotent embryonal carcinoma cells N2102Ep.

Biochimica et biophysica acta

Sutiwisesak R, Kitiyanant N, Kotchabhakdi N, Felsenfeld G, Andrews PW, Wongtrakoongate P.
PMID: 25086345
Biochim Biophys Acta. 2014 Nov;1843(11):2611-9. doi: 10.1016/j.bbamcr.2014.07.013. Epub 2014 Jul 30.

Embryonal carcinoma (EC) cells, which are considered to be malignant counterparts of embryonic stem cells, comprise the pluripotent stem cell component of teratocarcinomas, a form of testicular germ cell tumors (GCTs). Nevertheless, many established human EC cell lines are...

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Sutiwisesak R, Kitiyanant N, Kotchabhakdi N, et al. Induced pluripotency enables differentiation of human nullipotent embryonal carcinoma cells N2102Ep. Biochim Biophys Acta. 2014;1843(11):2611-9doi: 10.1016/j.bbamcr.2014.07.013.
Sutiwisesak, R., Kitiyanant, N., Kotchabhakdi, N., Felsenfeld, G., Andrews, P. W., & Wongtrakoongate, P. (2014). Induced pluripotency enables differentiation of human nullipotent embryonal carcinoma cells N2102Ep. Biochimica et biophysica acta, 1843(11), 2611-9. https://doi.org/10.1016/j.bbamcr.2014.07.013
Sutiwisesak, Rujapope, et al. "Induced pluripotency enables differentiation of human nullipotent embryonal carcinoma cells N2102Ep." Biochimica et biophysica acta vol. 1843,11 (2014): 2611-9. doi: https://doi.org/10.1016/j.bbamcr.2014.07.013
Sutiwisesak R, Kitiyanant N, Kotchabhakdi N, Felsenfeld G, Andrews PW, Wongtrakoongate P. Induced pluripotency enables differentiation of human nullipotent embryonal carcinoma cells N2102Ep. Biochim Biophys Acta. 2014 Nov;1843(11):2611-9. doi: 10.1016/j.bbamcr.2014.07.013. Epub 2014 Jul 30. PMID: 25086345.
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Corrigendum to "Review - Mitochondria-rich cells as experimental model in studies of epithelial chloride channels" [Biochim. Biophys. Acta 1566 (2002) 28-43].

Biochimica et biophysica acta

Willumsen NJ, Amstrup J, Møbjerg N, Jespersen Å, Kristensen P, Larsen EH.
PMID: 27289423
Biochim Biophys Acta. 2016 Sep;1858(9):2245-2246. doi: 10.1016/j.bbamem.2016.05.017. Epub 2016 Jun 09.

No abstract available.

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Willumsen NJ, Amstrup J, Møbjerg N, et al. Corrigendum to "Review - Mitochondria-rich cells as experimental model in studies of epithelial chloride channels" [Biochim. Biophys. Acta 1566 (2002) 28-43]. Biochim Biophys Acta. 2016;1858(9):2245-2246doi: 10.1016/j.bbamem.2016.05.017.
Willumsen, N. J., Amstrup, J., Møbjerg, N., Jespersen, �. �., Kristensen, P., & Larsen, E. H. (2016). Corrigendum to "Review - Mitochondria-rich cells as experimental model in studies of epithelial chloride channels" [Biochim. Biophys. Acta 1566 (2002) 28-43]. Biochimica et biophysica acta, 1858(9), 2245-2246. https://doi.org/10.1016/j.bbamem.2016.05.017
Willumsen, Niels J, et al. "Corrigendum to "Review - Mitochondria-rich cells as experimental model in studies of epithelial chloride channels" [Biochim. Biophys. Acta 1566 (2002) 28-43]." Biochimica et biophysica acta vol. 1858,9 (2016): 2245-2246. doi: https://doi.org/10.1016/j.bbamem.2016.05.017
Willumsen NJ, Amstrup J, Møbjerg N, Jespersen Å, Kristensen P, Larsen EH. Corrigendum to "Review - Mitochondria-rich cells as experimental model in studies of epithelial chloride channels" [Biochim. Biophys. Acta 1566 (2002) 28-43]. Biochim Biophys Acta. 2016 Sep;1858(9):2245-2246. doi: 10.1016/j.bbamem.2016.05.017. Epub 2016 Jun 09. PMID: 27289423.
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Post-transcriptional regulation of cardiac sodium channel gene SCN5A expression and function by miR-192-5p.

Biochimica et biophysica acta

Zhao Y, Huang Y, Li W, Wang Z, Zhan S, Zhou M, Yao Y, Zeng Z, Hou Y, Chen Q, Tu X, Wang QK, Huang Z.
PMID: 26209011
Biochim Biophys Acta. 2015 Oct;1852(10):2024-34. doi: 10.1016/j.bbadis.2015.07.016. Epub 2015 Jul 21.

The SCN5A gene encodes cardiac sodium channel Nav1.5 and causes lethal ventricular arrhythmias/sudden death and atrial fibrillation (AF) when mutated. MicroRNAs (miRNAs) are important post-transcriptional regulators of gene expression, and involved in the pathogenesis of many diseases. However, little...

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Zhao Y, Huang Y, Li W, et al. Post-transcriptional regulation of cardiac sodium channel gene SCN5A expression and function by miR-192-5p. Biochim Biophys Acta. 2015;1852(10):2024-34doi: 10.1016/j.bbadis.2015.07.016.
Zhao, Y., Huang, Y., Li, W., Wang, Z., Zhan, S., Zhou, M., Yao, Y., Zeng, Z., Hou, Y., Chen, Q., Tu, X., Wang, Q. K., & Huang, Z. (2015). Post-transcriptional regulation of cardiac sodium channel gene SCN5A expression and function by miR-192-5p. Biochimica et biophysica acta, 1852(10), 2024-34. https://doi.org/10.1016/j.bbadis.2015.07.016
Zhao, Yuanyuan, et al. "Post-transcriptional regulation of cardiac sodium channel gene SCN5A expression and function by miR-192-5p." Biochimica et biophysica acta vol. 1852,10 (2015): 2024-34. doi: https://doi.org/10.1016/j.bbadis.2015.07.016
Zhao Y, Huang Y, Li W, Wang Z, Zhan S, Zhou M, Yao Y, Zeng Z, Hou Y, Chen Q, Tu X, Wang QK, Huang Z. Post-transcriptional regulation of cardiac sodium channel gene SCN5A expression and function by miR-192-5p. Biochim Biophys Acta. 2015 Oct;1852(10):2024-34. doi: 10.1016/j.bbadis.2015.07.016. Epub 2015 Jul 21. PMID: 26209011; PMCID: PMC4757900.
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Proteomics profiling of cholangiocarcinoma exosomes: A potential role of oncogenic protein transferring in cancer progression.

Biochimica et biophysica acta

Dutta S, Reamtong O, Panvongsa W, Kitdumrongthum S, Janpipatkul K, Sangvanich P, Piyachaturawat P, Chairoungdua A.
PMID: 26148937
Biochim Biophys Acta. 2015 Sep;1852(9):1989-99. doi: 10.1016/j.bbadis.2015.06.024. Epub 2015 Jul 03.

Cholangiocarcinoma (CCA), a common primary malignant tumor of bile duct epithelia, is highly prevalent in Asian countries and unresponsive to chemotherapeutic drugs. Thus, a newly recognized biological entity for early diagnosis and treatment is highly needed. Exosomes are small...

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Dutta S, Reamtong O, Panvongsa W, et al. Proteomics profiling of cholangiocarcinoma exosomes: A potential role of oncogenic protein transferring in cancer progression. Biochim Biophys Acta. 2015;1852(9):1989-99doi: 10.1016/j.bbadis.2015.06.024.
Dutta, S., Reamtong, O., Panvongsa, W., Kitdumrongthum, S., Janpipatkul, K., Sangvanich, P., Piyachaturawat, P., & Chairoungdua, A. (2015). Proteomics profiling of cholangiocarcinoma exosomes: A potential role of oncogenic protein transferring in cancer progression. Biochimica et biophysica acta, 1852(9), 1989-99. https://doi.org/10.1016/j.bbadis.2015.06.024
Dutta, Suman, et al. "Proteomics profiling of cholangiocarcinoma exosomes: A potential role of oncogenic protein transferring in cancer progression." Biochimica et biophysica acta vol. 1852,9 (2015): 1989-99. doi: https://doi.org/10.1016/j.bbadis.2015.06.024
Dutta S, Reamtong O, Panvongsa W, Kitdumrongthum S, Janpipatkul K, Sangvanich P, Piyachaturawat P, Chairoungdua A. Proteomics profiling of cholangiocarcinoma exosomes: A potential role of oncogenic protein transferring in cancer progression. Biochim Biophys Acta. 2015 Sep;1852(9):1989-99. doi: 10.1016/j.bbadis.2015.06.024. Epub 2015 Jul 03. PMID: 26148937.
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Mechanism of aggregation and membrane interactions of mammalian prion protein.

Biochimica et biophysica acta. Biomembranes

Ambadi Thody S, Mathew MK, Udgaonkar JB.
PMID: 29514050
Biochim Biophys Acta Biomembr. 2018 Sep;1860(9):1927-1935. doi: 10.1016/j.bbamem.2018.02.031. Epub 2018 Mar 04.

The cellular prion protein (PrP

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Ambadi Thody S, Mathew MK, Udgaonkar JB. Mechanism of aggregation and membrane interactions of mammalian prion protein. Biochim Biophys Acta Biomembr. 2018;1860(9):1927-1935doi: 10.1016/j.bbamem.2018.02.031.
Ambadi Thody, S., Mathew, M. K., & Udgaonkar, J. B. (2018). Mechanism of aggregation and membrane interactions of mammalian prion protein. Biochimica et biophysica acta. Biomembranes, 1860(9), 1927-1935. https://doi.org/10.1016/j.bbamem.2018.02.031
Ambadi Thody, Sabareesan, et al. "Mechanism of aggregation and membrane interactions of mammalian prion protein." Biochimica et biophysica acta. Biomembranes vol. 1860,9 (2018): 1927-1935. doi: https://doi.org/10.1016/j.bbamem.2018.02.031
Ambadi Thody S, Mathew MK, Udgaonkar JB. Mechanism of aggregation and membrane interactions of mammalian prion protein. Biochim Biophys Acta Biomembr. 2018 Sep;1860(9):1927-1935. doi: 10.1016/j.bbamem.2018.02.031. Epub 2018 Mar 04. PMID: 29514050.
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